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This quantitative immunohistochemical investigation shed light on the dynamic cellular processes that contribute to the formation and progression of AP

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We sought to test the hypothesis that inhibition of the CD38 ecto-enzymatic activity using a heavy-chain monoclonal antibody Ab68 will, via augmenting organismal NAD + , prevent fibrosis in a mouse model of SSc characterized by NAD + depletion

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(h) a nucleotide sequence encoding an antigenic fragment of a HGPRBMY14 related polypeptide having an amino acid sequence encoded by a human cDNA in a cDNA plasmid contained in the ATCC deposit and described in Table I