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Tuberculosis Immune Reconstitution Inflammatory Syndrome: Profile of an Enigmatic Condition

In acute myeloid leukemia cells, reduced MTHFR function, whether due to polymorphisms or pharmacologic inhibition, decreases intracellular SAM, leading to loss of the repressive histone marks H3K27me3 and H3K9me3 and de-repression of transcription factors such as SPI1 (85)