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Mechanistically, the expression of GLS1 is regulated indirectly by Myc (via repression of miR-23a and miR-23b) and mTORC1 11,12

In vitro , the overexpression of this miRNA reduced migration and invasion of osteosarcoma cells transfected with a miRNA mimic

Our findings uncover an inflammatory, metabolically reprogrammed eosinophil population that promotes chronic skin inflammation by limiting protective T cell responses

By integrating clinical data, molecular biology evidence, and innovative model systems (e.g., bone marrow organoids), this study will provide a theoretical foundation for reversing ineffective erythropoiesis