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GHK-Cu as an anti-inflammatory agent Numerous studies have shown that GHK-Cu may reduce inflammation and free radical damage, repair protective skin barrier proteins, and improve the rate of healing following tissue injury [1]

To overcome this constraint, CNS-directed strategies often employ receptor-mediated transcytosis through engineered formats such as bispecific antibodies or brain-shuttle constructs targeting BBB receptors (e.g., TfR or insulin receptor) together with pathological targets such as A or tau, thereby improving brain delivery compared with conventional antibodies (Terstappen et al., 2021)

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This excitotoxicity is not limited to cortical areas but also occurs in subcortical structures like the IC, where salicylate-induced neuronal hyperactivity and oxidative injury are closely linked to NMDA receptor-mediated mechanisms (Wang et al., 2023)

Effects of pineal peptide preparation Epithalamin on free-radical processes in humans and animals